Findings
- Motion degraded study with abnormal T2/STIR signal hyperintensity in the cord and likely mild cord expansion extending inferiorly from the level of T7
- Multilevel small disc bulges without significant spinal canal stenosis
- Facet hypertrophy contributes to multilevel mild to moderate neural foraminal stenosis, most advanced bilaterally at T10-T11
- Vertebral venous malformations in the T7-T11 vertebral bodies
- Partially imaged aortic dissection extending from the aortic arch through the descending thoracic aorta in to the upper abdominal aorta
Diagnosis
Spinal cord infarct
Sample Report
Motion degraded study with abnormal T2/STIR signal hyperintensity in the cord and likely mild cord expansion extending inferiorly from the level of T7, which is particularly concerning for spinal cord infarct given reported paraplegia in the setting of an aortic dissection.
Multilevel small disc bulges without significant spinal canal stenosis. Facet hypertrophy contributes to multilevel mild to moderate neural foraminal stenosis, most advanced bilaterally at T10-T11.
Partially imaged aortic dissection extending from the aortic arch through the descending thoracic aorta in to the upper abdominal aorta, which can be further evaluated with aorta protocol CTA.
Discussion